Ramsay Hunt Syndrome: Use of corticoids? Case presentation

Authors

  • Luis Marcelo Aguirre Padilla Centro de Salud Guayzimi
  • Carlos David Rojas Delgado Hospital Básico de Amaluza
  • Cristian Patricio Muñoz Cevallos Puesto de Salud Chinapintza

Keywords:

Ramsay Hunt Syndrome, Herpes Zoster Otic, Facial paralysis.

Abstract

Ramsay Hunt Syndrome is produced by the reactivation of varicella zoster virus in the geniculate ganglion, affecting the VII cranial nerve and causing facial paralysis, vesicular injuries, radiculoneuropathy and ganglionitis. It represents 16% of all causes of unilateral facial paralysis in children and 18% in adults. The reactivation can be induced by stress, fever, radiotherapy, tissue damage, immunosuppression or idiopathically. The virus replicates along the dorsal roots of the sensory nerves, including the geniculate ganglion, and the facial nerve, causing painful ganglionitis. The complete clinical form associates an infectious and sensitive syndrome (otodinia and rash), which may be accompanied by ipsilateral facial neuropathy. We report the case of a 24-year-old patient with a history of varicella-zoster virus primoinfection at age 9, with a unilateral otalgia, vesiculobullous lesions in the facial region and left atrium, homolateral peripheral facial paralysis; with clinical evidence he is diagnosed with Ramsay Hunt Syndrome. In the management of this pathology, diagnosis and early treatment are fundamental, and although the single or combined therapy associating antivirals and corticosteroids is still controversial, the combined treatment demonstrated better results and fewer complications.

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Published

2017-12-31

How to Cite

Aguirre Padilla, L. M., Rojas Delgado, C. D., & Muñoz Cevallos, C. P. (2017). Ramsay Hunt Syndrome: Use of corticoids? Case presentation. CEDAMAZ, 7(1). Retrieved from https://revistas.unl.edu.ec/index.php/cedamaz/article/view/376

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Section

Research Articles

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